Home

Beporoz növényt A fedélzeten Kilencig tap73 is a marker of glutamine addiction in medulloblastoma Csiszoló rúd összeg

The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma  cells - Varon - 2019 - International Journal of Cancer - Wiley Online  Library
The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma cells - Varon - 2019 - International Journal of Cancer - Wiley Online Library

Beyond glucose: alternative sources of energy in glioblastoma
Beyond glucose: alternative sources of energy in glioblastoma

Enforced expression of Ebf3 in medulloblastoma CSCs results in enhanced...  | Download Scientific Diagram
Enforced expression of Ebf3 in medulloblastoma CSCs results in enhanced... | Download Scientific Diagram

Regulation of cancer cell metabolism: oncogenic MYC in the driver's seat |  Signal Transduction and Targeted Therapy
Regulation of cancer cell metabolism: oncogenic MYC in the driver's seat | Signal Transduction and Targeted Therapy

The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma  cells - Varon - 2019 - International Journal of Cancer - Wiley Online  Library
The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma cells - Varon - 2019 - International Journal of Cancer - Wiley Online Library

PDF) TAp73 is a marker of glutamine addiction in medulloblastoma
PDF) TAp73 is a marker of glutamine addiction in medulloblastoma

Molecules | Free Full-Text | Identification of Therapeutic Targets for  Medulloblastoma by Tissue-Specific Genome-Scale Metabolic Model
Molecules | Free Full-Text | Identification of Therapeutic Targets for Medulloblastoma by Tissue-Specific Genome-Scale Metabolic Model

TAp73 is a marker of glutamine addiction in medulloblastoma
TAp73 is a marker of glutamine addiction in medulloblastoma

TAp73 is a marker of glutamine addiction in medulloblastoma
TAp73 is a marker of glutamine addiction in medulloblastoma

TAp73 is a marker of glutamine addiction in medulloblastoma
TAp73 is a marker of glutamine addiction in medulloblastoma

Brain tumour's 'addiction' to common amino acid could be its weakness
Brain tumour's 'addiction' to common amino acid could be its weakness

Brain Tumor's 'Addiction' To Common Amino Acid Could Be Its Weakness
Brain Tumor's 'Addiction' To Common Amino Acid Could Be Its Weakness

Molecules | Free Full-Text | Identification of Therapeutic Targets for  Medulloblastoma by Tissue-Specific Genome-Scale Metabolic Model
Molecules | Free Full-Text | Identification of Therapeutic Targets for Medulloblastoma by Tissue-Specific Genome-Scale Metabolic Model

Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through  hexosamine biosynthetic pathway in pancreatic cancer | SpringerLink
Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreatic cancer | SpringerLink

Orally bioavailable glutamine antagonist prodrug JHU-083 penetrates mouse  brain and suppresses the growth of MYC-driven medulloblastoma -  ScienceDirect
Orally bioavailable glutamine antagonist prodrug JHU-083 penetrates mouse brain and suppresses the growth of MYC-driven medulloblastoma - ScienceDirect

TAp73 is a marker of glutamine addiction in medulloblastoma. - Abstract -  Europe PMC
TAp73 is a marker of glutamine addiction in medulloblastoma. - Abstract - Europe PMC

Frontiers | Targeting Glutaminolysis: New Perspectives to Understand Cancer  Development and Novel Strategies for Potential Target Therapies
Frontiers | Targeting Glutaminolysis: New Perspectives to Understand Cancer Development and Novel Strategies for Potential Target Therapies

Frontiers | Pathological implications of metabolic reprogramming and its  therapeutic potential in medulloblastoma
Frontiers | Pathological implications of metabolic reprogramming and its therapeutic potential in medulloblastoma

Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through  hexosamine biosynthetic pathway in pancreatic cancer | SpringerLink
Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreatic cancer | SpringerLink

Immunometabolism: A 'Hot' Switch for 'Cold' Pediatric Solid Tumors: Trends  in Cancer
Immunometabolism: A 'Hot' Switch for 'Cold' Pediatric Solid Tumors: Trends in Cancer

TAp73 is a marker of glutamine addiction in medulloblastoma. - Abstract -  Europe PMC
TAp73 is a marker of glutamine addiction in medulloblastoma. - Abstract - Europe PMC

Glutamine metabolism, the Achilles heel for medulloblastoma tumor | Cell  Death & Disease
Glutamine metabolism, the Achilles heel for medulloblastoma tumor | Cell Death & Disease

Frontiers | Pathological implications of metabolic reprogramming and its  therapeutic potential in medulloblastoma
Frontiers | Pathological implications of metabolic reprogramming and its therapeutic potential in medulloblastoma

NPI-0052 and γ-radiation induce a synergistic apoptotic effect in  medulloblastoma | Cell Death & Disease
NPI-0052 and γ-radiation induce a synergistic apoptotic effect in medulloblastoma | Cell Death & Disease

PDF) TAp73 is a marker of glutamine addiction in medulloblastoma
PDF) TAp73 is a marker of glutamine addiction in medulloblastoma

Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through  hexosamine biosynthetic pathway in pancreatic cancer | SpringerLink
Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreatic cancer | SpringerLink

The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma  cells - Varon - 2019 - International Journal of Cancer - Wiley Online  Library
The long noncoding RNA TP73‐AS1 promotes tumorigenicity of medulloblastoma cells - Varon - 2019 - International Journal of Cancer - Wiley Online Library